10.1093/ajcn/nqaa221 [DOI] [PubMed] [Google Scholar] Doets E
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1 Introduction Alzheimers disease (AD) is a neurodegenerative disorder characterized by progressive cognitive decline, memory loss, and neuropsychiatric symptoms, representing one of the most critical global public health challenges of the 21st century ( In this context, ferroptosisa newly defined, regulated cell death modality driven by iron-dependent lipid peroxidationhas rapidly emerged as a frontier and focus in AD research ( Ferroptosis, a regulated form of cell death, plays a significant role in various pathological conditions, including neurodegeneration, cancer, and ischemiareperfusion injury ( Figure 1 ) ( Figure 1 This review aims to systematically elucidate the central role of iron dyshomeostasis and the ferroptosis process it drives in the pathogenesis of Alzheimers disease
When there is a severe G6PD deficiency in the extra-embryonic tissues, the placenta development stops and the embryo dies 14
The hydrophilic portion of the A42 peptide coordinately forms a bond with transition metal ions such as copper (II), resulting in abnormal yet stable neurotoxic aggregates of A [5]
