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Regarding other ferroptosis-regulating systems than iron metabolism, we cannot exclude that glucocorticoids also affect the lipidome, which might contribute to the phenotype observed

28,29 Kornienko et al

Nitrosylation or glutathionylation of PDI leads to enzyme inactivation, activation of the unfolded protein response (UPR), and cancer cell death

MNX1-AS1 promotes phase separation of IGF2BP1 to drive c-Myc-mediated cell-cycle progression and proliferation in lung cancer

Role of NAC in Non-acetaminophen Poisonings The antidote effects of NAC to other toxic compounds are less well established than those to acetaminophen, and this is an area of toxicology still in development [9]

While NAPQI protein binding has also been observed at therapeutic doses in vitro, GSH depletion is still regarded as a pre-requisite for hepatotoxicity of APAP