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Through integrated proteomic and metabolomic analyses, we identified a high-inflammation, high-oxidation, low-metabolism triad in the hippocampus of CLP-induced SAE mice, providing novel insights into mechanisms underlying persistent cognitive deficits in SAE survivors

Thus, p53-induced apoptosis in response to (cyclic) fasting may cause recruitment and/or proliferation of LAMs

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10.1016/j.pneurobio.2010.08.001 162 WangN.ZengG.-Z.YinJ.-L.BianZ.-X