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BPC-157's action profile in treating side effects caused by NSAIDs is based on protein phosphorylation, inhibition of protein denaturation, stabilization of lysosomal and cellular membranes, inhibition of complement activation, inhibition of proteases and protein kinases, and fibrinolytic activity

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[8] Tethering the peptide to a long-lived plasma protein dramatically slows its clearance, extending its action from minutes to days

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Previous studies have demonstrated that elevated tension inhibited AT2 to AT1 differentiation and induces abnormal accumulation of pre-alveolar type I transitional cell state (PATS) (marked by the increased expression of KRT8 and CLDN4, Fig