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Targeting BTK with FDA-approved anticancer drugs, such as acalabrutinib and ibrutinib, which are metabolized by the CYP3A enzyme, common to both fungi and humans, can be combined with fungal morphogenesis inhibitors to enhance the apoptotic effect [183]

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By modulating the FAK-paxillin signaling cascade, BPC-157 may enhance cellular adhesion and promote more organized tissue reconstruction at the molecular level

Pan-cancer analyses reveal cancer-type-specific fungal ecologies and bacteriome interactions

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However, we cannot compare these results directly as kinetic results of copper release and incubation times, necessary for reaching the equilibrium were not presented in the earlier paper and it was only declared that the metal release from CuHSA in the presence of NTA is faster than in case of EDTA 28