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: Different mechanisms

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In general, the expression of HSP70 has become a major interest of studies because of its role in modulating inflammatory immune response and cytoprotection under stress conditions in a wide diversity of experimental injury models [73, 109]

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It now appears that RIP1 expression was found to be significantly increased in cartilage from OA patients and rat models of OA, together with in vivo evidence pointing to intra-articular RIP1 overexpression being sufficient to trigger OA symptoms in rats (176178)

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