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Vakifahmetoglu H, Olsson M, Zhivotovsky B

10.1002/hep.28251 21

At best, about half of all patients become unresponsive to their current therapy and need to be switched up to another, more toxic drug

Wnt5a potentiates TGF-beta signaling to promote colonic crypt regeneration after tissue injury

Since degradation of GSH and Gln by GGT from gastric helicobacters largely contribute to the glutamate-generating metabolism, we investigated the effect of GGT substrate supplementation (Gln/GSH) on the outcome of an experimental H