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Targeting metabolic checkpoints such as PKM2 or succinate oxidation can increase the availability of redox-active metabolites that accelerate lipid peroxidation in tumor cells

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doi: 10.3390/antiox9060518 189 MeftahiG

PERK phosphorylates the eukaryotic translation initiator factor 2 (eIF2), which increase the translation of transcription factor-4 (ATF4) and subsequently upregulating the expression of C/EBP homologous protein (CHOP) and tribbles homolog 3 (TRIB3)