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After administration of CB839, an inhibitor of GLS1, Th1 cells showed increased histone methylation, which resulted in decreased expression of PIK3IP1, a regulator of mammalian target of rapamycin complex 1 (mTORC1) signaling, and sensitivity to IL-2 signaling in Th1 cells, activating mTORC1 to promote the differentiation of effector Th1 cells ( Figure 2 ) (10)

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