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Upregulation of iron transport proteins such as transferrin (Tf), transferrin receptor (TfR), and divalent metal transporter 1 (DMT1) enhances cellular iron uptake, while loss or dysfunction of ferroportin 1 (FPN1) hampers iron efflux, leading to intracellular iron overload and oxidative damage

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The overexpression of an Amaranthus hypochondriacus NFYC gene modifies growth and confers water deficit stress resistance in Arabidopsis

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