A phase 2a, randomized, double-blind 28-day study of TZP-102 a ghrelin receptor agonist for diabetic gastroparesis
Thus, it is plausible that a local rise in endogenous FA might alter intracellular GSH leading to redox imbalance
In 2009, it was reported that TMAO degraded misfolded G98R mutant CRYAA by activating the ubiquitin-proteasome pathway, alleviating ERS and apoptosis of human lens epithelial B3 cells induced by aggregated G98R CrYAA
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[DOI] [PubMed] [Google Scholar] Cristofori P., Sauer A.V., Trevisan A
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