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Inhibition of VDAC1 function, as occurs via oligomerization blockade using DIDS, confers profound resistance to PTX by impairing mitochondrial apoptotic signaling, even in the presence of Bcl-2 inhibitors, like ABT-737 32

2g and Supplementary Fig 2f)

To start, compute the first and second derivative of with respect to , Since , there is potentially an inflection point at

Research on this is ongoing

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FIGURE 8 3.8 Immunohistochemical findings The immunohistochemical investigation demonstrated a significant increase in expression of proapoptotic makers, Bax, and caspase-3, in MTX treated group, which was associated with decreased antiapoptotic marker, Bcl-2, expression when compared to the CTRL group (Figure 9)