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thaliana GSH2 is the target of several redox PTMs that could impact its activity and thus modulate glutathione biosynthesis, i.e., sulfenylation (Wei et al., 2020) and nitrosylation (Figure 3 and Supplementary Table 4

Counteraction of perforated cecum lesions in rats: effects of pentadecapeptide BPC 157, L-NAME and L-arginine

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Additionally, urinary excretion of glutathione and cysteine surged significantly by 300-fold and ten-fold, respectively within just 90 minutes [7]

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