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However, Serum bilirubin concentrations mild increases, such as those in patients with Gilberts syndrome (GS, a benign form of unconjugated hyperbilirubinemia), as well as levels in the upper quartile of the currently accepted physiologic serum bilirubin range, have been reported to be negatively correlated with a wide range of disorders within the human body

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In another article, GA was shown to alter TGF-/Smad signaling pathway, thereby modifying dimethylnitrosamine-induced liver fibrosis in rats (Hussein et al., 2020)