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Casual observation during treatments showed that HFM appeared to exhibit mild cytotoxicity effects to external H 2 O 2 around the 250 uM concentration and levels above this would produce mass cell death/apoptosis in cultures within 24 h of treatment

These results, together with a series of complementary experiments, confirmed that the non-canonical activity of GCLC protects cystine-starved cells from ferroptosis in a GSH-independent manner 12

has demonstrated that it reduces myocardial hypertrophy and fibrosis in ventricular remodeling (93)

NBDHEX binds the GSTP1-1 H-site and forms a complex with GSH, inactivating the enzyme (Figs

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Of late, multiple transcription factors such as TFAP2C, p53, AP-1, STAT, Nrf2, HIF-1, and C/EBP have been linked with the transcriptional regulation of seleno-GPxs [8, 21,22,23,24]