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found that circ_0000808 was upregulated in NSCLC cells, where it enhanced the expression of the Gln transporter SLC1A5 by interacting with miR1827, thereby promoting Gln metabolism and facilitating the progression of NSCLC (56)
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Although the magnitude of hydraulic resistance experienced by migrating breast cancer cells in vivo is currently unknown, mathematical modeling indicates that depending on the interstitial fluid viscosity and matrix permeability, the hydraulic resistance within 3D matrices could potentially match or even exceed that observed in microchannels [50, 203]
Changes in heme levels during acute vaso-occlusive crisis in sickle cell anemia
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