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Whether this unique pattern worsens the obese state or constitutes a favorable adaptation remains unclear, and the physiological and pathophysiological implications of the altered GH/IGF-I axis in obesity remain uncertain with GH neither being the cause nor the solution to reverse obesity

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These tightly interconnected processes affect multiple KEs in the AOPN and underscore the need to integrate both genetic and epigenetic mechanisms in the assessment of lung cancer development under combined exposures

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