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De la Torre [7] has estimated that a treatment that delays the onset of symptoms by 5 years would reduce the number of AD patients by upwards of 50%

demonstrated that ZDHHC5-mediated palmitoylation of beclin 1 serves as a core mechanism for maintaining neuronal autophagy, and its deficiency in AD murine models impairs autophagic function, exacerbating proteotoxic deposition, thereby leading to neurodegeneration, long-term potentiation (LTP) deficits, and memory deficits (167)

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Comparison with Protein Biologics Relative to larger protein drugs, peptides often offer advantages in manufacturing, stability, and delivery

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