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The interpretation of these transcriptomic findings has been a continuing area of discussion, with some researchers viewing the broad effects as evidence of a regulatory function and others viewing them with caution given the difficulty of distinguishing direct effects from secondary consequences in such broad gene-expression landscapes

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The loss of the cartilage ECM is a key early feature of OA, further suggesting that the senescence of chondrocytes and other synovial joint cells is a driving factor in the pathogenesis of OA

L., Castanon-Cervantes, O., Evans, J

References 1.Kirnaz S et al