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GSH can be exhausted via inhibiting SLC7A11 by traditional drugs (e.g., sorafenib and sulfasalazine (SAS)) or novel compounds (e.g., erastin), thus inducing ferroptosis (Zhang et al., 2018), tumor protein 53 (TP53) (Jiang et al., 2015), nuclear factor E2 related factor 2 (NFE2L2/Nrf2) (Sun et al., 2016)

Future work should advance in three directions

Fatty liver disease arises from an excessive build-up of fat within the liver, potentially leading to functional impairments and disruptions in its regular operations

Adult celiac disease (celiac sprue), pernicious anemia and IgA deficiency

J Comput Chem 31(4):671690 White AC, Thannickal VJ, Fanburg BL (1994) Glutathione deficiency in human disease

Peng et al., 2021)