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The rate of treatment discontinuation due to adverse events was comparable to what has been observed in trials of GLP-1 monotherapy

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The review also covered three therapeutic research strategies: direct GSH depletion (leading to ferroptosis or enhanced chemotherapy sensitivity), indirect approaches targeting the tumour microenvironment, and prodrug strategies utilising elevated tumour GSH for drug activation

By blocking NF-B, it reduces the production of pro-inflammatory cytokines (IL-1, TNF-, IL-6) and inflammatory mediators (PGE2, NO) in keratinocytes and dermal cells

doi: 10.3390/ijms232415660 15 AngelovaD