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The acute hemolysis, that is the only common clinical manifestation associated with pathogenic variants in the G6PD gene, can rapidly be compensated for and thus, may remain undetectable

42 of the aforementioned 491 are located in their protein precursors conserved structural domains and they could be characterized by the presence of a secondary structure

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Conditions such as Parkinson's disease, Alzheimer's disease, multiple sclerosis, and stroke have all been associated with elevated levels of oxidative stress

This resonates strongly with our recent European Heart Journal study (Baka et al., EHJ 2025), where metabolic modulation with SGLT2 inhibition improved energetic efficiency, contractile balance, and hypertrophy