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The working hypothesis was straightforward: if biological ageing reflects a gradual decline in these endogenous peptide signals, then reintroducing them under controlled research conditions might offer insight into the mechanisms of cellular ageing itself

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Studies have shown that methionine-enriched diets or acute methionine loads cause homocysteine to be exported from cells, leading to increased plasma homocysteine levels (Ueland and Refsum, 1989)