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HGF/c-Met signaling is a well-established oncogenic pathway: c-Met is amplified or overexpressed in multiple cancers (gastric, lung, renal, hepatocellular) HGF/c-Met activation promotes tumor cell proliferation, survival, invasion, and metastasis c-Met inhibitors are FDA-approved cancer drugs (cabozantinib, crizotinib, capmatinib) A compound that potentiates HGF/c-Met signaling raises theoretical concerns about promoting tumor growth or transformation with chronic use

These antioxidants support glutathione recycling and enhance its effectiveness

Brian Salvas, Pharm.D

doi: 10.1016/j.bbadis.2018.08.012 257 KaserSMoschenACayonAKaserACrespoJPons-RomeroFet al

Patients who require corticosteroids for other medical conditions face additional challenges in fracture healing, and this is one scenario where teriparatide may be particularly beneficial

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