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McInnes IB, Schett G (2011): The pathogenesis of rheumatoid arthritis

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10.1038/ncb3064 41 FuW.MaY.LiL.LiuJ.FuL.GuoY.et al (2020)

6.2 Based on metabolic reprogramming Trained cells not only enhance gene expression via epigenetic remodeling but also reprogram metabolic pathways to meet elevated energy demands

In an in vitro model of Erastin-induced ferroptosis in PC12 neuronal cells, the iron inhibitor curcuminpolydopamine nanoparticles (Cur-PDA NPs) markedly enhanced the chelation of Fe 3+ and Fe 2+ , upregulated superoxide dismutase (SOD) and GSH expression, reduced Fe 2+ and ROS levels, inhibited lipid peroxidation, restored mitochondrial function, and attenuated neuronal ferroptosis