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In Vitro: Dihexa binds with high affinity to HGF and both dihexa and its parent compound Norleucine 1-AngIV induce c-Met phosphorylation in the presence of subthreshold concentrations of HGF and augment HGF-dependent cell scattering. Further, dihexa and Nle1-AngIV induce hippocampal spinogenesis and synaptogenesis similar to HGF itself. Dihexa effectively inhibits HGF dimerization at 1 M. While dihexa at 1 nM and 10 pM alone does not activate c-Met, it markedly augments the capacity of HGF at 1.25 and 2.5 ng/mL to activate c-Met

With this, the BPC 157 lowers the risk for diseases like atherosclerosis [1]

Martinez-Brito D, de la Torre X, Botr F

This efficient and effective method of treatment offers a powerful tool for maintaining and improving overall health

Suitability: Injections suit diagnosed deficiency

Muscle Preservation Sarcopenia (age-related muscle loss) often leads to weakness and reduced recovery