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, the covalent binding of NAPQI to the thiol groups of critical cellular proteins (following glutathione depletion), oxidative stress induced directly by NAPQI or via coproduction of reactive oxygen species (superoxide radical and hydrogen peroxide) through redox cycling between the paracetamol semiiminoquinone radical and NAPQI) (1, 2), and vice versa, these processes counteraction may be involved in BPC 157 beneficial effects

Ex vivo photothermal treatment-induced immunogenic cell death for anticancer vaccine development

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Phil Trans r Soc b 364:20972113

10.3390/nu11102296 45