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albicans exacerbates intestinal inflammation by inducing host production of pro-inflammatory mediators, including IL-1, and its abundance correlates with disease activity

This results in small differences in the overall yield of dsNNTTR between experiments

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Kainic acid-mediated excitotoxicity as a model for neurodegeneration

Neutrophils (purple) and macrophages (green) trigger an initial acute inflammatory response, phagocytosing dying endometriotic cells and tissue debris