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3 Interactions of autophagy with the cGAS-STING pathway 3.1 cGAS The interplay between autophagy and cGAS occurs through (i) cGAS-Beclin-1 interaction that promotes autophagic degradation of cytosolic dsDNA, (ii) cGAS-LC3 interaction that promotes micronucleophagy, and (iii) degradation of cGAS through p62-mediated selective autophagy ( Figure 3 )

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Aging-related RAS activation and vascular changes increase susceptibility to neurodegeneration, which Ang-II stimulating AHMs may effectively counteract, offering enhanced neuroprotection in this group 39

Conventional gastroprotective drugs commonly prescribed to minimize NSAID toxicity or heal NSAID-induced ulcers include H2-receptor antagonists (cimetidine, ranitidine, famotidine), proton pump inhibitors (omeprazole, lansoprazole, esomeprazole), and misoprostol (a synthetic prostaglandin analog) (Vonkeman 2010)

Using seasonal autoregressive integrated moving average methods, the model was trained and tested

active lesions are enriched in collagen-producing cells, aggregates of fibronectin and collagen, mesenchymal perivascular aggregates of platelet-derived growth factor receptor (PDGFR)-bearing cells, and pro-fibrotic factors such as biglycan and decorin