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Tirzepatide's dual mechanism allows for a higher dose ceiling because the GIP component modulates the gastrointestinal effects that would otherwise limit dosing

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Interventions, such as the addition of microRNAs, were tested on these organoids, showing promise in restoring the natural balance between neurons and glial cells

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Although the extent of BAD activation caused by GSB-106 was less prominent compared to BDNF, these findings still point out GSB-106 ability to mimic the neurotrophin-specific anti-apoptotic mechanism mediated by BAD protein