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Metformin, an antidiabetic agent, promotes glutamine metabolic dependency in PCa cells by restricting glucose-derived carbon entry into the TCA cycle, thereby forcing PCa cells to utilize glutamine reductive carboxylation for citrate production

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EVs isolated from BUTE cells were similar in size and morphology to those observed in vivo

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Matek, D., Matek, I., Staresinic, E., Japjec, M., Bojanic, I., Blagaic, A

Two randomized controlled trials, three prospective papers, one systematic review, and three clinical reviews fulfilled inclusion criteria