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To determine whether HFD altered gene expression in lesions established from WT endometrial tissues similar to that found in Klf9 -null lesions, we evaluated the expression of genes involved in estrogen receptor ( Esr1 and Esr2 ) and progesterone receptor ( Pgr-T, Pgr-B, Klf9, Dkk1 , and Notch-1 ) signaling pathways in lesions of recipients

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This explains why intracellular oxidation kinetics for roGFP2 fusion constructs with glutaredoxin mutants showed excellent correlation with the second order rate constants of the oxidative half-reaction from in vitro experiments 15,26,34,35

We found that ADH5 prevented the FA-dependent oxidation of Grx1-roGFP2 in the cytosol (Fig

Mitmesser, S