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longum BB 536 demonstrated to modulate the expression of TJPs, with the increase of zonulin-1 and 2, occluding and claudin-1, and to prevent inflammatory damage, in an in vitro model of intestinal barrier developed using Caco-2 cell monolayer [14]

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1A) but rather increased the expression of pNRF2 within these cells as measured by the amount of nuclear area occupied by pNRF2 (S

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Increased levels of tau(1/2) leu-enkephalin when levels were lower than normal in patients with generalized anxiety disorder and neurasthenia

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