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Importantly, genes such as Marco, involved in foam cell formation and matrix degradation[57, 58], Csf3, reported to be significantly increased in human AAA tissues[59, 60], Chil1, shown to be involved in vSMC phenotype switching [61, 62] and Olfm4, involved in pro-inflammatory response [63, 64] were uniquely downregulated in Mlkl / -d3-PPE animals

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Nrf2-deficient female mice develop lupus-like autoimmune nephritis

10.1007/s12603-015-0547-3 7 BakerJ.LiuJ.-P.RobertsonE

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