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Deletion of the APOCIII gene in knockout rabbits attenuates cholesterol diet-induced hyperlipidemia and protects against atherosclerosis

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C3a, along with another fragment called C5a that is generated from the cleavage of C5 by C5 convertases, plays a significant role in inflammation and immune modulation by robustly attracting inflammatory cells (C3a and C5a are also called anaphylotoxins) ( Table 1 ) that ensure that complement activity is precisely controlled, such that there is balancing of the need for a rapid response to invaders all while preserving/protecting the hosts cells ( Table 1 Complement System Regulators

Williams RR, Horm JW: Association of cancer sites with tobacco and alcohol consumption and socioeconomic status of patients: interview study from the Third National Cancer Survey

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