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A senolytic peptide called FOXO4-D-Retro-Inverso (FOXO4-DRI) has been reported to disrupt the interaction between FOXO4 and p53, allowing p53 to translocate to the cytosol and induce apoptosis

EGCG disrupts the enzyme needed for folate activation

This is in line with the fact that the processes driving R 2 relaxation arise from interactions between nuclear spins within the molecule and are sensitive to slower motions, such as internal motions (e NOEs, R 1 , and R 2 relaxation rates for p53 194 in presence and absence of FOXO4-DRI

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Once initial improvement is achieved, maintenance sessions may be recommended at longer intervals