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Six months later, the discolouration diminished

Under oxidative stress, excessive ROS emissions lead to mitochondrial damage, and loss or defects in mitochondrial energy production can enhance metabolic processes in the tricarboxylic acid (TCA) cycle, ETC, exacerbating ROS release and reducing antioxidant enzymes [81,82,83]

Many of these trials are extending the duration of treatment beyond initial feasibility studies, sometimes lasting 30 to 36 weeks, to evaluate sustained effects