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Several studies collectively support the hypothesis that redox dysregulation is central to ALS pathogenesis, particularly in genetically predisposed individuals

Remarkably, two major regulators of these pathways of phenotypic transition are somehow related to cholesterol metabolism, including APOE, involved in cholesterol extracellular transport, and TREM2, which has been recently shown to regulate the intracellular movement of cholesterol in microglia [42]

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"Improvement of alveolar glutathione and lung function but not oxidative state in cystic fibrosis." Am J Respir Crit Care Med

Journal of Biological Chemistry, 1994