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In stress response regulation, aerobic exercise acts as a key modulator exerting pivotal effects through the SESN family: it upregulates SESN1 expression, whose exercise-induced elevation activates AMPK and inhibits mTORC1 to reduce hepatic lipid synthesis/accumulation while enhancing fatty acid oxidation

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For a broader explanation of how research peptides function in scientific studies, read our Ultimate Guide to Research Peptides