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However, the primary purpose of antiviral drugs is not to regulate the hosts immune response but to only temporarily suppress virus replication

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To create the glutathione model used in this paper, we took the model from [11] and added the glutathione pathway, homocysteine, cystathionine, cysteine, glutamylcysteine, GSH, and GSSG [Figure 1] from a previous model of glutathione metabolism [14]

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