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The impact of glutathione depletion extends deeply into the realm of immune hypersensitivity, particularly concerning mast cell activation syndrome (MCAS)

OMM proteins such as NIX, FUNDC1, and BNIP3 can act as mitophagy-related receptors, relying on their LIR domain to bind with LC3, thereby directing dysfunctional mitochondria to autophagosomes for degradation via receptor-mediated mitophagy [71]

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HTY has also been suggested to influence genes and proteins involved in microbial growth and survival, which may further contribute to antimicrobial effects

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