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Amini, R

Redox Biology, 26, 101280

References Neelakantan H, Vance V, Wetzel MD, et al

RD16/0017/0012)

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In PEM patients, this delayed restoration of high-energy phosphate pools may indicate a fundamental deficit in mitochondrial respiratory function rather than a simple depletion of fuel reserves, and instead may point to more fundamental impairments in excitation-contraction coupling