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Up-regulation of n-Myc leads to overexpression of RPL15, sustained over-activation of ribosome biogenesis, and selective enhancement of -catenin, which may be involved in tumorigenesis of offspring of oxidative damage embryos through cross-interactions with Wnt and TGF-1 pathways [52, 55]

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Figure S3: Serial changes in the concentration of (A) GSH and (B) GSSG in diabetic subjects younger than 55 years