Petrovic, M
All applications restricted to controlled laboratory environments
Oxidative stress is a major by-product of cellular metabolism and its regulation is critical for preventing disease and aging
The growing understanding of AD as a multifactorial disease necessitates an approach that simultaneously addresses tau pathology, neuroinflammation, oxidative stress, mitochondrial dysfunction, and synaptic loss (Chu et al., 2024)
Im pulling this out of my having read Daniel Steinbergs papers
GLP-1s work through systemic signaling: brain appetite circuits, gut motility, pancreas, liver-facing metabolic changes, and for tirzepatide/retatrutide, additional incretin and glucagon-receptor effects